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Cellular and synaptic phenotypes lead to disrupted information
Fmr1-KO mice demonstrate enhanced responding upon rule reversal in the
NMD abnormalities during brain development in the Fmr1-knockout
Systemic pharmacological suppression of neural activity reverses
Isoform-selective phosphoinositide 3-kinase inhibition ameliorates
Cell type-specific disruption of cortico-striatal circuitry drives
Frontiers Parallel learning and cognitive flexibility
The activation of mGluR4 rescues parallel fiber synaptic
FMRP has a cell-type-specific role in CA1 pyramidal neurons to
α-Tocopherol Protects Against Oxidative Stress in the Fragile X